Publications

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Complement associated microvascular injury and thrombosis in the pathogenesis of severe COVID-19 infection: A report of five cases

Model for AP and LP complement activation by SARS-CoV2, and its interaction with coagulation cascades

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Docked SARS CoV-2 Proteins within the Cutaneous and Subcutaneous Microvasculature and their Role in the Pathogenesis of Severe COVID-19

Co-expression of ACE2 and SARS CoV-2 capsid proteins with the cutaneous and subcutaneous microvasculature in a normal deltoid skin biopsy from a patient with fatal COVID-19 associated ARDS.

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The differing pathophysiologies that underlie COVID-19 associated perniosis and thrombotic retiform purpura: a case series

Comparative light microscopic findings and myxovirus resistance protein A (MXA) pattern of immunoreactivity in COVID-19-associated perniosis, idiopathic perniosis and COVID-19-associated thrombotic retiform purpura.

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Severe COVID-19: A multifaceted viral vasculopathy syndrome

Graphic representation of the two distinct mechanisms of severe COVID-19.

Livedoid and Purpuric Skin Eruptions Associated With Coagulopathy in Severe COVID-19

Anti-complement C5 therapy with eculizumab in three cases of critical COVID-19

Strong homology between SARS-CoV-2 envelope protein and a Mycobacterium sp. antigen allows rapid diagnosis of Mycobacterial infections and may provide specific anti-SARS-CoV-2immunity via the BCG vaccine

Concomitant calciphylaxis and COVID-19 associated thrombotic retiform purpura

Focal small bowel thrombotic microvascular injury in COVID-19 mediated by the lectin complement pathway masquerading as lupus enteritis.

A Covid-19 Patient with Complement-Mediated Coagulopathy and Severe Thrombosis

Weill Cornell Medicine Dermatopathology 1300 York Avenue
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